Pet Safety / Compounds / Anatoxin-a

Is Anatoxin-a safe for dogs and cats?

High risk for pets

Dogs are at significant risk from anatoxin-a and other cyanotoxin exposure during recreational activities near freshwater bodies experiencing cyanobacterial harmful algal blooms. Dogs that swim in or drink bloom-affected water can ingest lethal quantities; dogs that roll in or lick algal scum washed ashore are at particularly high risk because dried cyanobacterial mats retain potent toxin concentrations. Anatoxin-a poisoning in dogs presents with rapid onset (15–30 minutes) of hypersalivation, muscle rigidity, tremors, seizures, and respiratory failure; death can occur within 30–60 minutes of significant exposure. Numerous dog fatalities from cyanotoxin (primarily anatoxin-a and/or microcystin) exposure at HAB-affected freshwater sites have been reported in the US, UK, Australia, and other countries, with public health authorities issuing dog advisories during confirmed bloom events. Other cyanotoxins — particularly microcystins — co-occur in blooms and contribute to hepatotoxicity in addition to the acute neurotoxicity of anatoxin-a. ASPCA Animal Poison Control Center lists cyanobacterial toxins as a significant, rapidly lethal hazard for dogs. There is no specific antidote; atropine is ineffective (anatoxin-a is an nAChR agonist, not an organophosphate); treatment is supportive ventilation.

What is anatoxin-a?

The IUPAC name is 1-[(1R,6R)-9-azabicyclo[4.2.1]non-2-en-2-yl]ethanone.

Also known as: 1-[(1R,6R)-9-azabicyclo[4.2.1]non-2-en-2-yl]ethanone, Anatoxin A, Anatoxin I, Antx-A.

IUPAC name
1-[(1R,6R)-9-azabicyclo[4.2.1]non-2-en-2-yl]ethanone
CAS number
64285-06-9
Molecular formula
C10H15NO
Molecular weight
165.23 g/mol
SMILES
CC(=O)C1=CCCC2CCC1N2
PubChem CID
3034748

Risk for dogs

High risk

Dogs are at significant risk from anatoxin-a and other cyanotoxin exposure during recreational activities near freshwater bodies experiencing cyanobacterial harmful algal blooms. Dogs that swim in or drink bloom-affected water can ingest lethal quantities; dogs that roll in or lick algal scum washed ashore are at particularly high risk because dried cyanobacterial mats retain potent toxin concentrations. Anatoxin-a poisoning in dogs presents with rapid onset (15–30 minutes) of hypersalivation, muscle rigidity, tremors, seizures, and respiratory failure; death can occur within 30–60 minutes of significant exposure. Numerous dog fatalities from cyanotoxin (primarily anatoxin-a and/or microcystin) exposure at HAB-affected freshwater sites have been reported in the US, UK, Australia, and other countries, with public health authorities issuing dog advisories during confirmed bloom events. Other cyanotoxins — particularly microcystins — co-occur in blooms and contribute to hepatotoxicity in addition to the acute neurotoxicity of anatoxin-a. ASPCA Animal Poison Control Center lists cyanobacterial toxins as a significant, rapidly lethal hazard for dogs. There is no specific antidote; atropine is ineffective (anatoxin-a is an nAChR agonist, not an organophosphate); treatment is supportive ventilation.

Regulatory consensus

1 regulatory bodyhas classified Anatoxin-a.

AgencyYearClassificationNotes
WHOGuideline value of 30 μg/L in recreational watersFor cyanotoxin-related risk management in water quality

Regulators apply different standards of evidence — animal-data weighting, exposure-pattern assumptions, epidemiological power thresholds — which is why two scientific bodies can review the same data and reach different conclusions. The disagreement is the data.

Where pets encounter anatoxin-a

  • Industrial FacilitiesManufacturing plants, Chemical storage areas, Waste treatment sites
  • Occupational EnvironmentsFactories, Warehouses, Transportation vehicles

Safer alternatives

Lower-risk approaches that achieve a similar outcome to Anatoxin-a:

  • Avoidance (no chemical substitute)
    Trade-offs: Direct chemical substitution requires verification that the replacement does not introduce new hazards (regrettable substitution). Conduct full hazard assessment of proposed alternative before adoption.
    Relative cost: 1.2-2×

Frequently asked questions

Is anatoxin-a safe for pets?

Dogs are at significant risk from anatoxin-a and other cyanotoxin exposure during recreational activities near freshwater bodies experiencing cyanobacterial harmful algal blooms. Dogs that swim in or drink bloom-affected water can ingest lethal quantities; dogs that roll in or lick algal scum washed ashore are at particularly high risk because dried cyanobacterial mats retain potent toxin concentrations. Anatoxin-a poisoning in dogs presents with rapid onset (15–30 minutes) of hypersalivation, muscle rigidity, tremors, seizures, and respiratory failure; death can occur within 30–60 minutes of significant exposure. Numerous dog fatalities from cyanotoxin (primarily anatoxin-a and/or microcystin) exposure at HAB-affected freshwater sites have been reported in the US, UK, Australia, and other countries, with public health authorities issuing dog advisories during confirmed bloom events. Other cyanotoxins — particularly microcystins — co-occur in blooms and contribute to hepatotoxicity in addition to the acute neurotoxicity of anatoxin-a. ASPCA Animal Poison Control Center lists cyanobacterial toxins as a significant, rapidly lethal hazard for dogs. There is no specific antidote; atropine is ineffective (anatoxin-a is an nAChR agonist, not an organophosphate); treatment is supportive ventilation.

What products contain anatoxin-a?

Anatoxin-a appears in: Manufacturing plants (Industrial facilities); Chemical storage areas (Industrial facilities); Factories (Occupational environments); Warehouses (Occupational environments).

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Sources (3)

  1. WHO: Cyanobacterial Toxins — Anatoxin-a, nAChR Mechanism, VFDF History, Recreational Water Guideline (30 μg/L), Drinking Water Treatment, Freshwater Ecology, and Global HAB Epidemiology (2021 Background Document) (2021) — regulatory
  2. US EPA: Cyanotoxins in Drinking Water — Anatoxin-a, Cylindrospermopsin, Microcystins — Health Advisories, Treatment Technologies, Monitoring Guidance, and HAB Early Warning Systems (2019) (2019) — regulatory
  3. ASPCA Animal Poison Control Center: Cyanobacterial Toxins (Anatoxin-a, Microcystin) — Freshwater HAB Exposure in Dogs, Rapid-Onset Neurotoxicity, Hepatotoxicity, Clinical Presentation, and Case Reports (2022) (2022) — veterinary

Reference data, not professional advice. Aggregates publicly available regulatory and scientific data; not a substitute for veterinary, medical, legal, or regulatory advice. Why we built ALETHEIA →